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 Aberrant Cell Biology in Cystic Fibrosis Cells. Cell Biological Basis for Aberrant Function in Infection and Inflammation in Cystic Fibrosis Respiratory Epithelial Cells
Наименование:

Aberrant Cell Biology in Cystic Fibrosis Cells. Cell Biological Basis for Aberrant Function in Infection and Inflammation in Cystic Fibrosis Respiratory Epithelial Cells

автор Joseph F. издательство Книга по Требованию Медико-биологические дисциплины
Cтраниц 132        мягкая обложка       
Аннотация:The second aspect focuses on the excessive production of a molecule named Sialyl-Lewis X (Slx) in CF cells. The third aspect involves the discovery of attenuated polymeric IgA transcytosis in CF cells, which may contribute to excessive colonization of P. aeruginosa. Future directions addressing these aspects of the cell biology of CF are outlined. This book describes a novel cell biological basis contributing to aberrant innate immunity and inflammation in the cystic fibrosis (CF) respiratory tract. Slx acts as a receptor for neutrophils which cause excessive damage in the CF bronchial epithelia. The first aspect covered involves the basis for previously described hyperacidification of organelles in CF cells, and how this phenomenon can be pharmacologically corrected.
Автор: Joseph F.
Издательство: Книга по Требованию
Год:2009
Цена:
2384 руб
Нет в наличии
Поиск: Aberrant Cell Biology in Cystic Fibrosis Cells. Cell Biological Basis for Aberrant Function in Infection and Inflammation in Cystic Fibrosis Respiratory Epithelial Cells
НОВЫЙ ГОД СИМВОЛ 2013 ГОДАУКРАШЕНИЯ
КАРНАВАЛ НА НОВЫЙ ГОД РАСПРОДАЖА
Ортомолекулярная медицина Orthomol Устрание причин заболеваний. Ортомоль это новый метод для лечения и профилактики болезней. Orthomol - ортомолекулярная медицина.
Вплив граміцидину S на агрегацію тромбоцитів і стійкість мембран еритроцитів до...

Индукция прооксидантами ПОЛ в мембранах тромбоцитов и эритроцитов повышает, а его торможение б – токоферолом понижает степень и скорость дезагрегации первых и скорость и время полного гемолиза вторых под действием грамицидина S. Влияние ПОЛ на биологическую активность грамицидина S зависит от температуры и в области 300С - 360С минимально. В то же время Пол снижает энергию активации как дезагрегации тромбоцитов, так и гемолиза эритроцитов. Биологическая активность грамицидина S по отношению к эритроцитам зависит от содержания холестерина и отношения холестерин/фосфолипиды в мембранах. Повышенный уровень этого отношения у больных с сердечно-сосудистыми заболеваниями снижает гемолитическое действие антибиотика. Ключевые слова: грамицидин S, тромбоциты, эритроциты, липиды мембран, агрегация, дезагрегация, гемолиз, ПОЛ, г-облучение. SUMMARY Adeeb Khalaf Fadel Al-Amoush. I flue ce of gramicidi S o pla ele s fu c io al proper ies a d ery hrocy e membra e s abili y. – he ma uscrip . he hesis for degree of ca dida e of scie ce i biology o speciali y 03.00.13 – physiology of ma a d a imals. – V. . Karazi Kharkov a io al U iversi y, Kharkov, 2008. he hesis is dedica ed o research of i flue ce of gramicidi S (GS) o pla ele s aggrega io a d ery hrocy e membra e s abili y agai s hemolysis i heal hy do ors a d pa ie s wi h some hear a d blood vessels disorders. ime, co ce ra io a d empera ure depe de cies of he ligh passage a d op ical de si y were a alyzed i pla ele -rich plasma a d ery hrocy e suspe sio for i ac cells, af er г-radia io a d ac iva io of POL i bo h cell ypes a d i rela io wi h lipid co e of ery hrocy e membra e GS effec s were differe for i dividual pla ele s a d a d ADP-i duced pla ele aggrega es.
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