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 MOLECULAR MECHANISMS OF HIV-1 LATENCY. Nature and Impact of HIV-1 Integration Sites
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MOLECULAR MECHANISMS OF HIV-1 LATENCY. Nature and Impact of HIV-1 Integration Sites

автор Yefei H. издательство Книга по Требованию Органическая химия
Cтраниц 116        мягкая обложка       
Аннотация:However, eradication of the infection has not been achieved. Because of its extremely slow decay rate, the latent reservoir is a major barrier to curing HIV-1 infection. Dr. Han’s dissertation explored the molecular mechanisms underlying this unique nature of the infection in resting CD4+ T cells, starting from the first in vivo analysis of integration sites in resting CD4+ T cells to the novel discovery of the bidirectional regulation on integrated HIV-1 by the host gene transcriptional read-through. This state of latency allows HIV-1 to evade immune responses and antiretroviral drugs. Upon cellular activation, replication-competent viruses can be quickly released from the latent reservoir to rekindle the infection. HIV-1 can establish latent infection in a small pool of resting memory T cells in which the provirus is stably integrated into the host genome but not producing viral proteins. HAART can reduce viremia to below the clinical limit of detection. Advances in the treatment of HIV-1 infection with highly active anti-retroviral therapy (HAART) have greatly reduced mortality and changed this fatal infection into a chronic disease for many.
Автор: Yefei H.
Издательство: Книга по Требованию
Год:2009
Цена:
2007 руб
Нет в наличии
Поиск: MOLECULAR MECHANISMS OF HIV-1 LATENCY. Nature and Impact of HIV-1 Integration Sites
НОВЫЙ ГОД СИМВОЛ 2013 ГОДАУКРАШЕНИЯ
КАРНАВАЛ НА НОВЫЙ ГОД РАСПРОДАЖА
Ортомолекулярная медицина Orthomol Устрание причин заболеваний. Ортомоль это новый метод для лечения и профилактики болезней. Orthomol - ортомолекулярная медицина.
ВИЧ-инфекция

По своему строению он не отличался от ВИЧ, тоже убивал Т-хелперы. Но, в противоположность ВИЧ, в сыворотках больных отсутствовали антитела к последнему и ДНК обоих вирусов не были идентичны. Авторы обозначили новый вирус как HIV-2 (ВИЧ-2). Сыворотки больных при ВИЧ-2 реагировали с вирусом зеленых мартышек, поэтому Монтанье считает, что у них может быть общее происхождение. Сравнительное изучение геномов ВИЧ-1 и ВИЧ-2 показало, что в эволюционном плане ВИЧ-2 далеко отстоит от ВИЧ-1. Авторы высказывают предположение, что оба вируса существовали задолго до возникновения современной эпидемии СПИДа. В некоторых странах Западной Африки встречены типичные случая СПИДа при отсутствии антител к ВИЧ. Больных выделены ретровирусы, которые подобно ВИЧ-1, обладают сродством к CD4-хелперам и вызывают их гибель. ВИЧ-2 обнаруживается главным образом в Западной Африке. ВИЧ-2 родственен возбудителю СПИД-подобного заболевания у макак. Он способен инфицировать разные виды приматов, удаленные от человека на лестнице эволюции, тогда как ВИЧ-1 заражает только людей и шимпанзе.
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