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 A Molecular and Functional Approach Regulatory Mechanisms Controlling HIV-1 Induced Immune Activation.
Наименование:

A Molecular and Functional Approach Regulatory Mechanisms Controlling HIV-1 Induced Immune Activation.

автор Druzhkova S.G. издательство Книга по Требованию Общая биология
Cтраниц 76        мягкая обложка       
Аннотация:We used an SIV/pigtailed macaque model of HIV-1 disease to elucidate the role of Tregs in HIV-1-induced depletion of CD4+ T cells in lymphoid tissue during the acute phase of infection. The assays developed in our laboratory should be especially useful to the future studies of both virologists and immunologists. Using assays developed in the macaque model, we next studied the role of Tregs in elite suppressors who are HIV-1-infected individuals that maintain normal CD4+ T cell counts and control viremia without therapy. A specialized cell population, the T regulatory cells (Tregs), exists to help limit chronic inflammation by suppressing the immune activation induced by infectious diseases. The most widely accepted theory for the massive depletion of CD4+ T cells in HIV-1 infection involves depletion of non-infected cells through immune activation. From this model, we learned that Tregs play a significant role in controlling the apoptotic loss of CD4+ T cells resulting from high levels of generalized immune activation.
Автор: Druzhkova S.G.
Издательство: Книга по Требованию
Год:2008
Цена:
2007 руб
Нет в наличии
Поиск: A Molecular and Functional Approach Regulatory Mechanisms Controlling HIV-1 Induced Immune Activation.
НОВЫЙ ГОД СИМВОЛ 2013 ГОДАУКРАШЕНИЯ
КАРНАВАЛ НА НОВЫЙ ГОД РАСПРОДАЖА
Ортомолекулярная медицина Orthomol Устрание причин заболеваний. Ортомоль это новый метод для лечения и профилактики болезней. Orthomol - ортомолекулярная медицина.
ВИЧ-инфекция

По своему строению он не отличался от ВИЧ, тоже убивал Т-хелперы. Но, в противоположность ВИЧ, в сыворотках больных отсутствовали антитела к последнему и ДНК обоих вирусов не были идентичны. Авторы обозначили новый вирус как HIV-2 (ВИЧ-2). Сыворотки больных при ВИЧ-2 реагировали с вирусом зеленых мартышек, поэтому Монтанье считает, что у них может быть общее происхождение. Сравнительное изучение геномов ВИЧ-1 и ВИЧ-2 показало, что в эволюционном плане ВИЧ-2 далеко отстоит от ВИЧ-1. Авторы высказывают предположение, что оба вируса существовали задолго до возникновения современной эпидемии СПИДа. В некоторых странах Западной Африки встречены типичные случая СПИДа при отсутствии антител к ВИЧ. Больных выделены ретровирусы, которые подобно ВИЧ-1, обладают сродством к CD4-хелперам и вызывают их гибель. ВИЧ-2 обнаруживается главным образом в Западной Африке. ВИЧ-2 родственен возбудителю СПИД-подобного заболевания у макак. Он способен инфицировать разные виды приматов, удаленные от человека на лестнице эволюции, тогда как ВИЧ-1 заражает только людей и шимпанзе.
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